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In page Endoplasmic reticulum:
"The endoplasmic reticulum does not harbor an ATP-regeneration machinery, and therefore requires ATP import from mitochondria. The imported ATP is vital for the ER to carry out its house keeping cellular functions, such as for protein folding and trafficking.[1] The ER ATP transporter, SLC35B1/AXER, was recently cloned and characterized,[2] and the mitochondria supply ATP to the ER through a Ca2+-Antagonized Transport into the ER (CaATiER) mechanism [3] The CaATiER mechanism shows sensitivity to cytosolic Ca2+ ranging from high nM to low μM range, with the Ca2+-sensing element yet to be identified and validated.
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