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In page Integrin beta 6:
"A major driver of fibroblast activation is TGF-β[3] and as αvβ6 expression is increased in response to tissue damage,[4] and is a principal activator of TGF-β, it is therefore a potential drug target in treating fibrosis. αvβ6 can promote fibrosis in kidney, lung and skin, despite avb6 being almost absent in their healthy equivalents.
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